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I have essential thrombocytemia and have been on Hydrea for more than 5 years. The dosage differs from time to time, according to my blood tests. Actually I undergo blood tests every months since the time I was diagnosed with the disease. This is not the first thrombocytemia medication I am on, but this is by far the best one. With Hydrea I have least side effects and it provides excellent results on my platelets level. However I have some nausea and occasional headaches I feel much better with Hydrea than with any other med. I do hope that one day I will be able to find a more effective treatment that will help me conquer the illness but in general I am happy with Hydrea as it makes my quality of life so much higher. My doctor is also satisfied with the results we achieve when on Hydrea and he plans to continue this treatment course. I wish him lots of luck!)

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When I first started taking Hydroxyurea it made me a little sick. Yet, after about 2 weeks I noticed a slight increase in energy and went from having a hospitalization crisis every 2-3 months to about ONCE a year. I still take pain meds for the smaller pains but my lifestyle had gotten MUCH better. I would recommend it to any person with sickle cell.

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More recently, gluten intolerance was used by those both in and outside the medical community to describe the unpleasant symptoms that occur when people who do not have celiac disease nonetheless react to glutencontaining foods.

hydroxyurea may improve lung function in children with sickle cell disease

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Bone marrow testing is also important in monitoring the progress of the disease, particularly during the spent phase.

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Of the 38 genes that interacted genetically with CNB1, 23 were hypersensitive to both FK506 and CsA and a further 5 were hypersensitive to FK506 alone.

What are the possible late side effects of treatment with hydroxyurea?

Nonetheless, the evidence in children does not contradict the findings in adults that hydroxyurea improves hematologic variables and decreases hospitalization rates.

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Impact of hydroxyurea on clinical events in the BABY HUG trial.

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It is also used to reduce the frequency of painful sickle cell crisis.

Hydroxyurea Safety Trial, often referred to as HUG-KIDS.

Engage and support community-based efforts to improve knowledge of the benefits and risks of hydroxyurea.

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Because of the potential for serious adverse reactions with hydroxyurea, a decision should be made whether to discontinue nursing or to discontinue the drug, taking into account the importance of the drug to the mother.

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Observational studies in both adults and children support the use of hydroxyurea in reducing the complications of sickle cell disease (including pain, hospitalizations, blood transfusions, and acute chest syndrome) and decreasing mortality.

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CPR alone cannot stand as the sole important link to increased survival, as a significant barrier to successful CPR is the complexity of the skills required.

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Financial Support: Support for the development of this guideline was provided by the AABB, Bethesda, Maryland.

In a report on a sickle cell workshop, reviewed current treatment of this disease.

It was also shown, in vitro, that hydroxyurea reacts directly with deoxy-sickle-cell hemoglobin to form methemoglobin and nitrosyl hemoglobin (HbNO).

Hyroxyurea exhibits increased effectiveness when used in conjunction with irradiation therapy.

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Due to potentially severe clinical outcomes for the cutaneous vasculitic ulcers reported in patients with myeloproliferative disease, hydroxyurea should be discontinued if cutaneous vasculitic ulcerations develop and alternative cytoreductive agents should be initiated as indicated.

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Fetal hemoglobin (HbF) is the major genetic modulator of the hematologic and clinical features of sickle cell disease, an effect mediated by its exclusion from the sickle hemoglobin polymer.

Eight percent of the patients (two women and seven men; median age, 78 years; range, 66 to 85) died of cardiac failure.

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Leukamias that develop over a long period of time come under the term chronic, the growth of the disorder is much slower and the symptoms are less dramatically felt, individuals affected by this form of leukamias can thus expect to lead much longer lives, even without any treatment.

With the necessary support, people with sickle cell disease should be able to lead normal lives.

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RNA repair may be a particularly appropriate genetic approach with which to treat sickle cell disease because the process should restore the regulated expression of anti-sickling versions of beta-globin and simultaneously reduce the production of sickle beta-globin.

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Hydroxyurea is excreted by the kidney, and the risk of adverse reactions to this drug may be greater in patients with impaired renal function.

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It can cause pain and weakness, and is typically caused by repetitive motions over a long period of time.

If the pain is new, especially when accompanied by more jaundice than usual, nausea and vomiting, then further hepatobiliary workup is needed.

Sickle cell disease (SCD) is characterized by intermittent vaso-occlusive events and chronic hemolytic anemia.

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We knew that data on the long-term harms of hydroxyurea in individuals with sickle cell disease would be limited.

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People who have sickle cell trait are protected from malaria and do not develop sickle cell disease, but they are “carriers” who can pass the abnormal gene on to their children.

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About 40% of males, including children, with sickle cell disease suffer from priapism.

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